Glucocorticoid secretion is thought to contribute to stress adaptation by causing long-term changes in gene expression via cognate adrenocorticosteroid
receptors (i.e., mineralocorticoid receptor [MR] and glucocorticoid receptor [GR]).
These physical responses are part of an interactive feedback loop that releases stress hormones such as norepinephrine, adrenocorticosteroid
hormone, and cortisol when persons experience stress.
Animal studies have shown that TBI is associated with hippocampal damage in CA3, DG, and, to a lesser degree, CA1  and that although the mechanisms leading to hippocampal damage in TBI are complex, it is partially mediated by posttraumatically increased adrenal steroids and altered adrenocorticosteroid
receptor properties in those subfields [201-202].
treatment of hydrocarbon pneumonia in children--a cooperative study.