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Inflammation of the periodontium.



an acute or chronic inflammation of the periodontium and adjacent tissues. Periodontitis is usually a result of dental caries and arises as infection spreads from the root canal through the apical foramen to the apex of the root. It may also develop from frequently recurring injury to the tooth; this kind of injury can result from such occupational habits as biting thread or grasping metal nails with the teeth and from any habitual biting of hard objects, for example, pencils or the mouthpieces of pipes.

Acute periodontitis is manifested by sharp pain in the region of the tooth that intensifies when the inflamed area is touched. Swelling often occurs in the gums, lips, or cheeks, and the affected tooth becomes loose. The enlarged submaxillary lymph nodes become sensitive, and fever occasionally arises. The process may sometimes be complicated by osteomyelitis of the jaw, purulent inflammation of the soft tissues of the face and neck, and acute sepsis. Symptoms of chronic periodontitis usually include discomfort while chewing, halitosis, and sometimes fistulas on the gums and facial skin. Chronic periodontitis can lead to the formation of a cyst of the jaw, and both forms of periodontitis can sensitize the body to streptococci.

Treatment of periodontitis is usually conservative, consisting of filling the root canals. Often the tooth is removed. Abscesses are lanced, and antibiotics are used to treat pronounced general symptoms.


Groshikov, M. I. Periodontit. Moscow, 1964.
Marchenko, A. I. “Bolezni periodonta.” In Rukovodstvo po terapevticheskoi stomatologii. Moscow, 1967.
Ovrutskii, G. D., F. G. Gasimov, and S. V. Makarov. Bolezni zubov. Kazan, 1967.
Rybakov, A. I., and V. S. Ivanov. Klinika terapevticheskoi stomatologii. Moscow, 1973.


References in periodicals archive ?
Aggressive periodontitis (AgP) is a rapidly progressing form of periodontal disease.
Immunologic and microbiologic profiles of chronic and aggressive periodontitis subjects.
demonstrate that in localized aggressive periodontitis patients, allele 2 of IL--1 RN VNTR(variable no.
Bozoglan, "Comparison of CCL28, interleukin-8, interleukin-1[beta] and tumor necrosis factor-alpha in subjects with gingivitis, chronic periodontitis and generalized aggressive periodontitis," Journal of Periodontal Research, vol.
05) (table 1) in both chronic periodontitis and aggressive periodontitis
Abstract: The aim was to assess the efficacy of antimicrobial photodynamic therapy (aPDT) in the treatment of aggressive periodontitis (AgP).
Subgingival microbiota in chronic and aggressive periodontitis in Bogota, Colombia: an epidemiological approach.
Serum levels of cytokines in subjects with generalized chronic and aggressive periodontitis before and after non-surgical periodontal therapy: A pilot study.
This case report describes the interdisciplinary management of four controlled aggressive periodontitis cases that were treated in a conservative manner.
Occurrence of Actinobacillus actinomycetemcomitans in patients with chronic periodontitis, aggressive periodontitis, healthy subjects and children with gingivitis in two cities of the state of Sao Paulo, Brazil.
1-q143 resulting in phagocytic particularly macrophage dysfunction thus reducing individual's immunity towards micro-organisms residing/flourishing in periodontal pockets causing aggressive periodontitis.
Prevalence of periodontopathic bacteria in aggressive periodontitis patients in a Chilean population.

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