alpha receptor


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Related to alpha receptor: beta receptor

alpha receptor

[¦al·fə ri¦sep·tər]
(cell and molecular biology)
Any of a group of receptors on cell membranes that are thought to be associated with vasoconstriction, relaxation of intestinal muscle, and contraction of the nictitating membrane, iris dilator muscle, smooth muscle of the spleen, and muscular layer of the uterine wall. Also called alpha-adrenergic receptor.
References in periodicals archive ?
Intrahepatic expression of interferon alpha and interferon alpha receptor m-RNA can be used as predictors to interferon response in HCV and HCC patients.
Association of Interferon Alpha Receptor 1 with sustained virological response in hepatitis C and B co-infected patients.
If the mice missing the alpha receptor still had a functional beta version, their bodies still could respond to estrogen.
Conclusion: Interferon alpha receptors 1-messenger ribonucleic acid may be useful for predicting response to interferon plus ribavirin therapy in hepatitis C virus/ hepatitis B virus co-infected patients who were females with lower hepatitis C virus-ribonucleic acid and hepatitis B virus-deoxyribonucleic acid levels.
These nine articles describe some of the work being done to differentiate markers and use their presence to detect and treat a range of cancers, addressing the characterization of breast cancer subtypes in a large retrospective study, intraocular lymphoma markers, polymorphisms in genes associated with certain T-cell lymphomas, tumor antigens that are markers of minimal residual disease in acute myeloid leukemia, thyroid tumor markers, activity and expression of dipeptidyl peptidase IV and cathepsin H in human cutaneous melanoma compared to other common cancers, melanoma inhibitory activity as a serological marker in metastic melanoma, and the promise (or myth) of the tumor necrosis factor alpha receptors p55 and p75 in ovarian cancer detection.
Alpha receptors cause body fat to be stored in our system, while beta receptors cause it to be released.
Wright explains that estrogen stimulation of alpha receptors tends to increase cell proliferation, while beta receptors tend to decrease it.
Alpha receptors trigger the cells to constrict coronary arteries, and the beta receptors have an opposing effect.