anaerobic glycolysis

anaerobic glycolysis

[¦an·ə¦rōb·ik glī′käl·ə·səs]
(biochemistry)
A metabolic pathway in plants by which, in the absence of oxygen, hexose is broken down to lactic acid and ethanol with some adenosinetriphosphate synthesis.
References in periodicals archive ?
Anaerobic power is the maximal power developed during all-out, short-term effort and reflects the energy-output capacity of intramuscular high-energy adenosine triphosphate and phosphocreatine (ATP and PCr) and/or anaerobic glycolysis. Anaerobic capacity is the maximum amount of ATP resynthesized via anaerobic metabolism during a specific mode of short-duration maximal exercise (Green and Dawson, 1993).
In addition, the accumulation of hypocotyls between the ascending epithelial cells of the anaerobic glycolysis end, lactate is first passed through the endothelium to the stroma.
Lactate is formed from the reduction of pyruvate, which is generated largely by anaerobic glycolysis. In tissue hypoxia, lactate is overproduced via increased anaerobic glycolysis and worsens if liver dysfunction and acute kidney injury occur due to a decreased ability to clear lactate from the body.
In the present study, HS-induced upregulation of lactate and pyruvate in the liver (p<0.01) pointed to increased anaerobic glycolysis. In common with this finding, lactate and pyruvate were also upregulated in blood and milk in heat-stressed dairy cows in our previous studies [4,5].
When oxygen is not available, they produce energy via the less efficient route of anaerobic glycolysis. In the 1920s, Otto Warburg observed that cancer cells do not produce energy in the efficient way that normal cells do.
GLUT-1 likewise provides glucose for cells to perform anaerobic glycolysis in hypoxic environments.
The results suggest that Ldh-c expresses in skeletal muscles of plateau pika, and the pika skeletal muscle may get at least 32.42% ATP for its activities by LDH-C4 catalyzed anaerobic glycolysis, which reduces the dependence on oxygen and enhances the adaptation to the hypoxic environments.
Dual metabolic regulation by long-term HSCs (LT-HSCs) during self-renewal and quiescence primarily relies on anaerobic glycolysis. Lineage-committed progenitors rapidly enter into the Krebs cycle to meet the unique set of bioenergetic demands required for cell proliferation and differentiation via the mitochondrial oxidative phosphorylation (OXPHOS) switch.
As per literature, aggressive tumor cells tend to divide quickly which outgrow their blood supply creating relative hypoxia within the tumor bed leading to anaerobic glycolysis with activation of enzyme lactate dehydrogenase and production of lactate.
Glucose is the predominant energy substrate that can be oxidized to C[O.sub.2] and [H.sub.2]O through tricarboxylic acid (TCA) cycle or transformed into lactate by anaerobic glycolysis. In this study, an increase in lactate level and a decrease in glucose level were found in the serum after A[[beta].sub.25-35] injection from 1 to 2 weeks, indicating that anaerobic glycolysis was upregulated during AD development.
Pyruvate is converted to lactate; that is, anaerobic glycolysis is activated.
The Warberg effect is the malignant cell's propensity to utilize anaerobic glycolysis over mitochondrion-dependent metabolism.