Autolysis

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autolysis

[ȯ′täl·ə·səs]
(geochemistry)
Return of a substance to solution, as of phosphate removed from seawater by plankton and returned when these organisms die and decay.
(pathology)
Self-digestion by body cells following somatic or organ death or ischemic injury.

Autolysis

 

self-digestion of animal, plant, and microorganism tissue. In autolysis the cellular proteins, carbohydrates, and fats break down under the action of hydrolytic enzymes in the cells. In living organisms, autolysis is evident in necrotic centers and in cells of malignant neoplasms. Autolysis occurs in decomposing corpses. In plants autolysis occurs after the death of living cells as a result of low temperature, desiccation, and the action of such poisonous substances as chloroform and toluene, as well as during mechanical tissue disintegration. Autolysis of microbe cells is evident in aging microbe cultures and in microorganisms damaged by physical, chemical, or biological agents. Autolysis also occurs in several industrial processes—for example, in tobacco and tea fermentation and during feed storage.

N. P. MESHKOVA

References in periodicals archive ?
Accumulation of gastric juice and pepsin which involves in the autodigestion of gastric mucosa is implicated in ulcer incidence in PL model (30).
The digestive enzymes elicit autodigestion of pancreatic tissue and a massive inflammatory response (Despins, Kivlahan, & Cox, 2005).
Such a diet not only provides wide spectrum composition of food but also because of its autodigestion characteristics, facilitates a better assimilation in larvae.
La Pancreatitis Aguda (PA) es una enfermedad inflamatoria, causada por la activacion, liberacion intersticial y autodigestion de la glandula por sus propias enzimas (1).
1) The pathophysiology is proposed to be autodigestion of the peripancreatic arterial wall secondary to released proteolytic enzymes or erosion of a pseudocyst into the artery and conversion of its cavity into a pseudoaneurysm.
It is evident from the above that alcohol exerts constant and toxic effects on different cellular compartments in the pancreas, predisposing the gland to autodigestion, necroinflammation, and fibrosis.