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Inflammation of the thyroid gland.



inflammation of the thyroid gland caused by nonspecific infection (for example, staphylococcosis), specific infection (tuberculosis), poisoning (by lead or carbon monoxide), or autoimmune diseases. Symptoms include pain upon swallowing or moving the head backward, throbbing pain in the ears and lower jaw, elevated body temperature, enlargement of the neck, and tenderness of the regional lymph nodes. The course of thyroiditis may be acute, subacute, or chronic. Treatment calls for the use of antiinflammatory agents, analgesics, antibiotics, corticosteroids, thyroidin, and vitamins. Suppurative thyroiditis is treated surgically.

References in periodicals archive ?
Sixty-two HT patients, 20 with euthyroid autoimmune thyroiditis, 27 with subclinical hypothyroidism and 15 with overt hypothyroidism constituted the patient groups.
Resistance to experimental autoimmune thyroiditis induced by physiologic manipulation of thyroglobulin level.
Central pontine myelinolysis (CPM), steroid-responsive encephalopathy associated with autoimmune thyroiditis (SREAT), Bickerstaff's brainstem encephalitis (BBE) are some of the disorders that might present with a localized lesion that specifically involves the pontine area (3).
Membranous nephropathy associated to autoimmune thyroiditis, chronic pancreatitis and suprarenal insufficiency [in Spanish].
Exceptions may apply to females with postpartum autoimmune thyroiditis.
There is a high prevalence of autoimmune thyroiditis in women with polycystic ovary syndrome, according to a prospective study presented at the annual meeting of the American Thyroid Association.
The study groups consisted of consecutive patients seen at our clinics for primary hypothyroidism based on autoimmune thyroiditis (n = 37; 10 males and 27 females; median age, 46 years; range, 22-72 years) and for hyperthyroidism caused by Graves disease (n = 14; 1 male and 13 females; median age, 41 years; range, 23-73 years).
Idiotypic/antiidiotypic interactions are suggested to suppress tissue damage caused by autoantibodies in a number of conditions including autoimmune thyroiditis, SLE, pernicious anemia, cold agglutinin disease, myasthenia gravis, and idiopathic thrombocytopenic purpura.
The primary outcomes tested whether dietary supplementation with anatabine ameliorates the immune system's targeting of the thyroid gland in cases of autoimmune thyroiditis.
However, patients suffering from disorders such as rheumatoid arthritis (RA), ankylosing spondylitis (AS), and autoimmune thyroiditis (i.