The researchers looked at
cardiac muscle cells -- cardiomyocytes (CMs).
Therefore, in the present study, we sought to identify an exercise method capable of effectively preventing cardiovascular diseases through protection of the
cardiac muscle. Specifically, we compared the effects between two 12-week exercise regimes (aerobic and resistance) on ER stress and mitochondrial biogenesis in the
cardiac muscle of middle-aged rats with high-fat-diet-induced obesity.
Measurement of Exhaustive Exercise-Induced Skeletal and
Cardiac Muscle Damage.
IR injury and inflammatory response can substantially activate cell autophagy [6], and autophagy has been demonstrated to play an important role in the pathologic process of
cardiac muscle injury [7].
These pressures, created by the contraction of
cardiac muscle in the ventricle walls, control the opening and closing of the semilunar valves and thus the movement of blood from the left and right ventricles into the aorta and pulmonary trunk.
The ventricular tissues of water-pipe exposed rats, showed some degree of separation between
cardiac muscle fibers, infiltration of lymphocytes, and congestion of blood vessel.
One of the reasons for this is the fact that
cardiac muscle cells do not regenerate.
*An impulse-conducting system of the heart for initiation and propagation of electrical impulses for
cardiac muscle contraction It is formed by highly specialized
cardiac muscle cells (Purkinje fibers), which generate and conduct electrical impulses rapidly through the heart.
Skeletal and
cardiac muscles are collectively referred to as striated muscle.
A team of Harvard University researchers has transplanted artificial
cardiac muscle cells developed from multipurpose stem cells into six heart-failure patients in the United States in the world's first clinical application of such cells, one of the researchers said Wednesday.
[1] So Serum enzyme AST and protein troponin I are the best indicators for
cardiac muscle injury because of their early rise, i.e.
Organization of 108 chapters is in sections on
cardiac muscle (basic physiology, adaptations and response, myocardial disease); skeletal muscle (basics and adaptations, disease, and therapeutics); and smooth muscle (physiology, heterogeneities, adaptations and response, and disease).