complementary genes


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Related to complementary genes: epistasis, Multiple alleles

complementary genes

[‚käm·plə′men·trē ′jēnz]
(genetics)
Nonallelic genes for which a wild type allele of one abolishes the phenotypic effect of a mutation of the other.
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Chi-squared (kh2) tests of the resistant and susceptible plants fitted a ratio of 1:63 (kh2 {1:63} =1.56 less than 3.84, P=0.10-0.25), confirming the result that the resistance in Early Premium to 2E16 was controlled by three recessive complementary genes. The two results are consistent, indicating the resistance was inherited by nucleus.
Chi- squared (kh2) tests of the resistant and susceptible plants fitted a ratio of 1:63 (kh2 {1:63} =1.30 less than 3.84, P=0.25-0.50), indicating that the resistance to CYR18 in Early Premium was controlled by three recessive complementary genes.
In recent work, we have built mathematical models of speciation that are explicitly based upon the accumulation of complementary genes (Orr 1993, 1995; Turelli and Orr 1995).
Use of tightly linked markers (Miklas et al., 1996; Urrea et al., 1996) for target genes found in different bean races should facilitate both determination of their genetic divergence and value for pyramiding complementary genes. Moreover, identification of molecular markers tightly linked to each of the other components of BGM resistance traits (Molina Castaneda and Beaver, 1998; Morales and Niessen, 1988; Velez et al., 1998) should also receive priority.
Segregation of RILs of CRIL-7 showed a good fit to the 1 resistant:7 susceptible ratio expected for three recessive complementary genes (Table 4).
The existence of two different C18:1 segregations for one or two complementary genes in crosses with CAS-3 (Table 2) suggested the presence of C18:1 dominant alleles in some genotypes of CAS-3, as reported previously for other low C18:1 lines (Fernandez-Martinez et al., 1989).
To accumulate complementary genes into one strain, two crossing designs can be used, single crossing and multiple crossing.
The observed distribution of 1 resistant : 3 susceptible lines indicated this IT X to be produced by a pair of complementary genes. Contingency analysis showed two chromosomal regions, on chromosome arms 3BS and 4BL (Fig.

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