Motor nerve conduction velocities (MNCVs) were not evoked in the distal lower limbs, while very low amplitudes of compound motor
action potential and mildly decreased MNCVs in the other motor nerves were recorded.
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There was also a positive trend on two novel biomarkers: rate of decline of muscle volume and of compound motor axon potential (CMAPs).
The investigators also looked at changes in muscle mass (estimated by 3 dimensional MRI) and in compound motor axon potential (CMAP) during the six month post treatment period compared with those in the run-in period.
Nerve conduction study showed diminished Compound Motor
Action Potential (CMAP) with normal conduction velocity in both median, ulnar and peroneal nerves.
Electrodiagnostic studies showed mildly reduced compound motor
action potential amplitudes and normal sensory responses.
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Electromyography demonstrated a trend of stabilization of the compound motor
axon potential in the right musculocutaneous nerve as compared to deterioration observed in the left.
Electrodiagnostic studies of all patients displayed markedly reduced compound motor
axon potentials with preserved sensory nerve action potentials; the results were interpreted as consistent with a process primarily localized to the motor axons or, far more likely, given the clinical scenario and pathologic data from WNV-infected animals, the anterior horn cells of the spinal cord.
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