The present work is mainly concerned with the study of various electrodiagnostic parameters, i.e., DML, motor nerve conduction velocity, and compound motor action potential amplitude in normal healthy adult population of Haryana.
1998  Present study Right side Moto 3.1[+ or -]0.3 2.9[+ or -]0.58 rmedian 11.1[+ or -]2.8 11.72[+ or -]5.22 56.5[+ or -]3.5 52.58[+ or -]6.62 Motor ulnar 2.4[+ or -]0.3 2.07[+ or -]0.44 9.2[+ or -]2.2 10.02[+ or -]3.22 60.4[+ or -]5.2 53.28[+ or -]6.62 Nerve Present study Left side Moto 2.88[+ or -]0.61 rmedian 12.32[+ or -]5.44 52.48[+ or -]5.92 Motor ulnar 2.11[+ or -]0.52 9.8[+ or -]2.88 52.18[+ or -]5.32 DML: Distal motor latency, CMAP: Compound muscle action potential, CMAPA: Compound motor action potential amplitude, MNCV: Motor nerve conduction velocity, CV: Conduction velocity
Motor nerve conduction velocities (MNCVs) were not evoked in the distal lower limbs, while very low amplitudes of compound motor
action potential and mildly decreased MNCVs in the other motor nerves were recorded.
There was also a positive trend on two novel biomarkers: rate of decline of muscle volume and of compound motor
axon potential (CMAPs).
Nerve conduction study showed diminished Compound Motor
Action Potential (CMAP) with normal conduction velocity in both median, ulnar and peroneal nerves.
Electrodiagnostic studies of all patients displayed markedly reduced compound motor
axon potentials with preserved sensory nerve action potentials; the results were interpreted as consistent with a process primarily localized to the motor axons or, far more likely, given the clinical scenario and pathologic data from WNV-infected animals, the anterior horn cells of the spinal cord.
Electromyography demonstrated a trend of stabilization of the compound motor
axon potential in the right musculocutaneous nerve as compared to deterioration observed in the left.