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Related to encephalopathies: alcoholic encephalopathy


Any disease of the brain.



a collective term that designates a noninflammatory organic disease of the brain. Some encephalopathies are innate, resulting from embryopathy. Others are the result of infection, poisoning, trauma, or vascular disease of the brain. There are no specific manifestations. The most common encephalopathies resemble neuroses (asthenia, irritability, insomnia, headaches) or psychoses (narrowed scope of interests, passivity, emotional instability, vulgarity). Symptoms may include memory loss or mental deterioration.

Alcoholic encephalopathies are alcoholic psychoses. They may be acute, as in Wernicke’s encephalopathy (named for the German neuropathologist C. Wernicke, who described the condition in 1881), or chronic, as in Korsakov’s psychosis (named for S. S. Korsakov) and alcoholic pseudoparalysis. Lead encephalopathy is caused by chronic poisoning by lead salts.

Treatment for encephalopathies depends on the cause of the disease.

References in periodicals archive ?
Beyond BSE: Transmissible spongiform encephalopathies in other animal species.
Eating meat contaminated with infectious prions is only one way that spongiform encephalopathies are transmitted.
The SSC eventually foresees setting up a system where livestock can be certified free of spongiform encephalopathies.
Rohwer, at the Cambridge Healthtech Institute's 10th Annual Transmissible Spongiform Encephalopathies Conference in Baltimore, MD.
Transmissible spongiform encephalopathies have perhaps the most unconventional natural history of any infectious agent.
At the request of the Food and Drug Administration (FDA) Transmissible Spongiform Encephalopathies (TSEs) Advisory Committee, Pall Corporation (NYSE: PLL) today presented the latest research results on its prion technology to remove TSE infectivity from blood.
Prion diseases, also known as transmissible spongiform encephalopathies, are rapidly progressive, uniformly fatal brain diseases that can infect humans and animals, including cattle, sheep, goats, mink, deer, elk, cats, and zoo ungulates.
The labeling states that the manufacture of Gamunex(R), which incorporates the innovative Caprylate/Chromatography process for improved product purity and prolonged biological activity, provides reasonable assurance that potential infection risk is significantly reduced from pathogenic prions associated with Transmissible Spongiform Encephalopathies (TSEs), such as vCJD, in the unlikely event they are present in donated plasma.
We present the results of mouse bioassay studies to show that, contrary to findings in cattle with BSE in which the tissue distribution of infectivity is the most limited recorded for any of the transmissible spongiform encephalopathies (TSE), infectivity in greater kudu with BSE is distributed in as wide a range of tissues as occurs in any TSE.
An update on Transmissible Spongiform Encephalopathies (TSEs), including variant Creutzfeldt-Jakob Disease (vCJD), the human form of "mad cow" disease, was a key topic at the Food & Drug Administration's Blood Products Advisory Committee meeting in Gaithersburg, Maryland today.
Such age specificity of disease risk may be a general feature of transmissible spongiform encephalopathies, which suggests that a general mechanism should be sought.