exocytosis


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exocytosis

[¦ek·sō·sī′tō·səs]
(cell and molecular biology)
The extrusion of material from a cell.
McGraw-Hill Dictionary of Scientific & Technical Terms, 6E, Copyright © 2003 by The McGraw-Hill Companies, Inc.
References in periodicals archive ?
In particular, syntaxin 1A has a role in the synaptic exocytosis process and neuronal plasticity (36).
Histopathology of allergic CD shows lymphocytic perivascular infiltrate, high dermal edema, and epidermal spongiosis and exocytosis in the dermis (2).
[58.] Cao P, Maximov A, Sudhof TC (2011) Activity-Dependent IGF-1 Exocytosis Is Controlled by the Ca2+-Sensor Synaptotagmin-10.
The mechanism of cell death is apoptosis via drug-induced CD8+ cell exocytosis of granzyme B/perforin and granulysin and through the activation of the Fas-Fas ligand pathway and tumor necrosis factor-alpha/death receptor pathway.
In neurons, deficiencies in priming molecules (such as Munc13s, a protein family implicated in vesicle priming/maturation before exocytosis), caused the RRP to be almost lost, whereas the number of morphologically docked vesicles remained intact.
Typical histological findings include spongiotic and lichenoid interface dermatitis with superficial and deep perivascular infiltrate and epidermal changes including hyperkeratosis, parakeratosis, focal spongiosis, and lymphocytic exocytosis. The deep lymphohistiocytic infiltrates are also seen surrounding adnexal structures such as hair follicles and eccrine glands [11-13].
Molecules, e.g., neurotransmitters and proteins, are released from the cell by exocytosis [1].
Skin punch biopsy revealed an ulcerative lesion with a fibrinous cap and spongiotic vesicles (Figure 4) with marked eosinophilic exocytosis associated with dyskeratotic keratinocytes and eosinophilic intraepidermal microabscess formation (Figure 5).
Upon nasal administration, NPL enter the cells by endocytosis and return to the lumen through exocytosis [13], without crossing the first epithelial cell layer [9].
catarrhalis OMVs towards the A549 airway epithelium, and no data are available on their influence on OMV-induced neutrophil granule exocytosis. Therefore, the present study was designed to elucidate the impact of both neuropeptides on (i) OMV-stimulated hBD-2 promoter activity in human A549 epithelial cells (type II alveolar cells) as constituents of the first line of defense, (ii) OMV-mediated A549 apoptotic response, and (iii) the azurophilic and specific granule release from neutrophils--the processes associated with the damage of surrounding tissues.
Biopsy from ileum showed moderate lymphocytic infiltration with lymphoid follicle formation in lamina propria and some glands in the rectum showed polymorphonuclears exocytosis that resulted in reparative dysplastic changes, destruction, goblet cell depletion, and crypt abscess formation (figure 2).