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The time rate of change of velocity. Since velocity is a directed or vector quantity involving both magnitude and direction, a velocity may change by a change of magnitude (speed) or by a change of direction or both. It follows that acceleration is also a directed, or vector, quantity. If the magnitude of the velocity of a body changes from v1 ft/s to v2 ft/s in t seconds, then the average acceleration a has a magnitude given by Eq. (1):
Instantaneous acceleration is defined as the limit of the ratio of the velocity change to the elapsed time as the time interval approaches zero. When the acceleration is constant, the average acceleration and the instantaneous acceleration are equal.
Whenever a body is acted upon by an unbalanced force, it will undergo acceleration. If it is moving in a constant direction, the acting force will produce a continuous change in speed. If it is moving with a constant speed, the acting force will produce an acceleration consisting of a continuous change of direction. In the general case, the acting force may produce both a change of speed and a change of direction.
Angular acceleration is a vector quantity representing the rate of change of angular velocity of a body experiencing rotational motion. If, for example, at an instant t1, a rigid body is rotating about an axis with an angular velocity ω1, and at a later time t2, it has an angular velocity ω2, the average angular acceleration α is given by Eq. (2),
When a body moves in a circular path with constant linear speed at each point in its path, it is also being constantly accelerated toward the center of the circle under the action of the force required to constrain it to move in its circular path. This acceleration toward the center of path is called radial acceleration. The component of linear acceleration tangent to the path of a particle subject to an angular acceleration about the axis of rotation is called tangential acceleration. See Rotational motion
(1) In biological evolution, an acceleration of the development of some parts of the embryo at a definite stage of development. The Russian scientist A. N. Severtsov used the term acceleration to describe a situation in which a particular organ appears earlier in the ontogenesis and develops faster than other organs; as a result this organ begins functioning when the conditions of life make it necessary. Examples of acceleration include the earlier evolution of the mouth apparatus of fish and all ecaudate amphibians, which makes possible their feeding after their reserves of yolk have been exhausted; the evolution of milk teeth among mammals; and changes in the nature of fins and the development of vertebral arches and ribs among some fish when these bone elements are formed, bypassing the stage of cartilaginous skeleton formations.
L. IA. BLIAKHER
(2) In anthropology the more rapid somatic development and physiological maturation of children in adolescence that has been observed in the past 100–150 years; in a wider sense it refers to changes in bodily measurements and rates of somatic development in the course of a man’s whole life cycle. Acceleration occurs among all social groups of the population and refers to a variety of anatomical and physiological indications. Sometimes the term psychological acceleration is used in the literature to refer to accelerated mental development, although there is as yet no reliable statistical material on the subject. Changes in bodily measurement have been better studied than other areas. The size of children at birth is on the average 0.5–1 cm greater and the weight 100–300 grams greater than 100 years ago. Between 1880 and 1950 the height of children ages 5–7 increased by 1.5 cm every 10 years and the weight by 0.5 kg. The height of school-age children rose 10–15 cm in the same period. The age of sexual maturation has dropped an average of from one to two years in the past 75–100 years. Thus, the average of the first menstruation in Europe dropped from 16.5 years in the early 19th century to 13–13.5 years. The climacteric sets on not at 43–45 years, as was the case in the early 20th century, but at 48–50 years.
There are many hypotheses on the causes of acceleration: better food, development of hygiene, the neurogenic hypothesis (which explains acceleration in big cities as the action of urban stimuli on the nervous system); and several endogenous hypotheses (for instance, one that explains acceleration through the greater number of marriages between people from distant places). But all these hypotheses are one-sided and open to criticism. Most likely, acceleration is caused by many biological and social factors. The significance of acceleration for mankind still has not been studied.
REFERENCESBunak, V. V. “Ob uvelichenii rosta i uskorenii polovogo sozrevaniia sovremennoi molodezhi v svete sovetskikh somatologicheskikh issledovanii.” Voprosy antropologii, 1968, issue 28.
Solovieva, V. S. “Obzor nekotorykh gipotez o prichinakh aktseleratsii.” Voprosy antropologii, 1967, issue 26.
Tanner, J. M. Growth at Adolescence. Oxford, 1962.
V. S. SOLOV’EVA and N. N. MIKLASHEVSKAIA
a vector quantity characterizing the rate at which the velocity of a point changes in magnitude and direction with respect to time. If the point is in rectilinear motion and its velocity v increases or decreases uniformly, the magnitude of the acceleration is w = Δv/Δt, where Δv is the change in velocity in the time interval Δt. In the general case the acceleration vector w is the first derivative of the velocity vector v with respect to time: w = dv/dt = v̇. The acceleration vector is directed inward on the concave side of the point’s path and lies in the osculating plane.
In a rectangular Cartesian coordinate system the projections of acceleration on the x-axis, y-axis, and z-axis are equal to the first derivatives of the projections of velocity, or to the second derivatives of the coordinates of the point, with respect to time: wx = v̇x – ẍ, wy = v̇y = ÿ, and wz = v̇z = z̈. Moreover, the absolute value of acceleration is
At each point on the path of motion the acceleration has a tangential component wT and a normal, or centripetal, component wn; the former is the projection of the acceleration on the tangent to the path, and the latter is the projection of the acceleration on the principal normal to the path. The two components are defined by the equations wτ = dv/dt = v̇ and wn = v2/ρ, where v is the magnitude of the velocity and ρ is the radius of curvature of the path at the point in question. It can be shown that
The tangential component characterizes the change in the magnitude of the velocity, and the normal component characterizes the change in the direction of the velocity.
The relation among the acceleration of a free material particle, the particle’s mass m, and the applied force F is given by Newton’s second law: mw = F. The dimensions of acceleration are LT–2.
The acceleration of points of a rotating body is discussed in ROTATIONAL MOTION OF RIGID BODIES and ANGULAR ACCELERATION.
S. M. TARG
Physiological effects. According to the character of the effects on an organism, three types of acceleration are distinguished: impact linear acceleration, here defined as acceleration having a duration of not more than 1 sec and a rate of onset of not less than 10 g/sec (1 g is the acceleration of gravity at sea level); sustained linear acceleration, here defined as acceleration lasting not less than 1 sec and having a rate of onset of not more than 10 g/sec; and angular acceleration. In aviation and space medicine the “increased weight” of the body owing to acceleration is characterized by what is called the g factor.
A person is subjected to the greatest impact linear accelerations in falls, transportation accidents, the emergency landing of an airplane or spacecraft, ejection, and so on. The principal adverse pathophysiological effect of impact acceleration is damage to organs and tissues (spine, skull, and internal organs). The tolerance to impact accelerations directed perpendicularly to the body’s long axis is approximately twice as great as the tolerance to accelerations directed along the long axis; the respective tolerances are 30–40 g and 15–20 g. In the course of evolution man developed specific mechanisms of protection against impact acceleration, such as the shock-absorbing properties of the bone-support apparatus and the suspension system of the internal organs.
The extent of the adverse effect of sustained linear acceleration depends on the magnitude of the acceleration and its direction relative to the person’s body. The more closely the acceleration
vector approaches the body’s long axis and the direction of the main blood vessels, the more pronounced are the disruptions of circulation associated with the redistribution of blood due to increased hydrostatic pressure. Accelerations that lead to an increase in the amount of blood in vessels in the head are tolerated most poorly. A person withstands sustained acceleration best when the acceleration vector forms an angle of 75°–80° with the long axis of the body (see Figure 1). This condition is met on spacecraft of the Soyuz and Apollo types. The greatest sustained linear accelerations that a person may be subjected to at the present time occur during the maneuvering of a high-speed airplane or during the flight of a spacecraft along a ballistic trajectory. In the course of evolution man had practically no encounters with sustained linear acceleration. The tolerance to such acceleration is determined by general, nonspecific mechanisms of adaptation to adverse factors in the environment.
Rotary motions involve angular accelerations, which exert a specific influence on the vestibular apparatus and at certain values may bring about effects characteristic of impact and sustained linear acceleration.
Tolerance to acceleration is increased through the use of various types of equipment to maintain the person’s optimal posture and position with respect to the acceleration vector, to lower the magnitude and rate of increase of the acceleration, and to reduce the effects of blood redistribution in the organism. Such equipment includes shock-absorbing, form-fitting seats, safety belts, crash helmets, and G suits.
REFERENCESBarer, A. S. “Problemy uskorenii v kosmicheskoi fiziologii.” Kosmicheskaia biologiia i medilsina, 1967, issue 1.
Sergeev, A. A. Fiziologicheskie mekhanizmy deistviia uskorenii. Leningrad, 1967.
Kralkii spravochnik po kosmicheskoi biologii i meditsine, 2nd ed. Moscow, 1972.
Osnovy kosmicheskoi biologii i medilsiny: Sovmesmoe sovetskoamerikanskoe izdanie, vol. 2, book 1. Moscow, 1975.
A. S. BARER
accelerator(1) See particle accelerator.
(2) Speeding up the retrieval of Web pages. See Web page acceleration and CDN.
(3) Speeding up file downloading. See download accelerator.
(4) Speeding up hardware. See graphics accelerator, video accelerator and accelerator card.
(5) A quick way to gain knowledge when reading text on a Web page. For example, after highlighting a word and clicking a button, an accelerator may provide a dictionary definition, a foreign language translation or the map of a street address.
(6) A key combination such as Alt-Shift-G that is used to activate a task. It provides a faster activation method than selecting from a menu. See also special function key.
(7) An incubator that expects to develop the company considerably faster than normal. See incubator.