Embryopathy

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Related to fetopathy: Fetation, macrosomia, diabetic fetopathy

embryopathy

[‚em·brē′äp·ə·thē]
(medicine)
Any abnormal development of an embryo, either morphological or biochemical.
McGraw-Hill Dictionary of Scientific & Technical Terms, 6E, Copyright © 2003 by The McGraw-Hill Companies, Inc.
The following article is from The Great Soviet Encyclopedia (1979). It might be outdated or ideologically biased.

Embryopathy

 

a disease of or injury to a human embryo occurring between the middle of the first and the end of the third month of intrauterine development. Embryopathy may be caused by a genetic disturbance or by a pathogenic factor that affects the embryo through the mother, such as hypoxia, poisoning, or an infectious disease. Embryopathy may result in malformation of embryonic organs, developmental anomalies, and spontaneous abortion. Prevention calls for the protection of the mother’s health during the first months of pregnancy.

The Great Soviet Encyclopedia, 3rd Edition (1970-1979). © 2010 The Gale Group, Inc. All rights reserved.
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We illustrate a case of angiotensin II receptor antagonistrelated fetopathy with mild symptoms in the neonatal period.
Quan, "Fetopathy associated with exposure to angiotensin converting enzyme inhibitors and angiotensin receptor antagonists," Early Human Development, vol.
All participating TISs used a standard form to report exposure details, delivery outcomes, and specific fetal or infant features associated with ACEI fetopathy. Renal function, growth retardation, and skull ossification defects were assessed.
(110-113) Angiotensin-converting enzyme inhibitor fetopathy results from direct effects on the renin-angiotensin system as well as a reduction in blood flow and oxygenation to the fetus.
Severe diabetic fetopathy despite strict metabolic control.
If the outcome variable is the health of the fetus and neonate, then the question should be, "What test identifies women at risk for diabetic fetopathy (macrosomia and neonatal metabolic aberrancy)?" (11).
Pregnant women with even mild hyperphenylalaninemia also require strict dietary control of Phe concentrations to prevent PKU-induced fetopathy (1).