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Dysplasia development is lower than 1% in sporadic fundic gland polyps and there is no relationship between atrophic gastritis and H.
All visible polyps were either biopsied or removed and thus identified and studied them histologically by the pathologist and classified them as adenomatous polyps, hyper-plastic polyps, fundic gland polyps, and other polyps.
In concordance to our observation, very high frequencies (71.3%) of hyperplastic polypi have also been reported in the recent literature.22 In divergence to our results, a few studies have also reported fundic gland polypi (FGP) to be more prevalent than hyperplastic polypi.
In this study, it was found that the distribution of endocrine cells was the most in the pylorus gland, the more in the fundic gland, and the less in the cardiac glands.
There was a little difference in different gland region, in fundic gland region mucosa which was highest, and in pyloric gland region was lowest;and in contents which was lowest because of milk and grass diluted (0.003 to 0.009 U/mg protein)(Table 5).
Areport of gastric fundic gland polyps, Gastroentrol Hepatol.
Parietal cells: In fact, the cells of the fundic glands with high succinic dehydrogenase activity are parietal cells at the 8th week of development (37).
pylori infection, the gastric mucosa showed closed- or open-type atrophy, but the gastric mucosa at the GIST site showed grade B0 gastritis in the fundic glands of 4 patients, grade B1-B3 gastritis in 18 patients, no grade A atrophy, and a grade A1 lesion in the antral pyloric glands of 1 patient.