glucose-6-phosphate dehydrogenase


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Related to glucose-6-phosphate dehydrogenase: pentose phosphate pathway, glucose-6-phosphate dehydrogenase test

glucose-6-phosphate dehydrogenase

[′glü‚kōs ¦siks ′fäs‚fāt dē‚hī′drä·jə‚nās]
(biochemistry)
The mammalian enzyme that catalyzes the oxidation of glucose-6-phosphate by TPN+(triphosphopyridine nucleotide).
McGraw-Hill Dictionary of Scientific & Technical Terms, 6E, Copyright © 2003 by The McGraw-Hill Companies, Inc.
References in periodicals archive ?
ciliata decreased the levels of tumor markers: aspartate aminotransferase, alanine aminotransferase, lactate dehydrogenase, alpha-fetoprotein, gamma glutamyl transferase, 5' nucleotidase and glucose-6-phosphate dehydrogenase. It was concluded that bioactive compounds B.
Severe neonatal hyperbilirubinemia: a potential complication of glucose-6-phosphate dehydrogenase deficiency.
Beutler, "Glucose-6-phosphate dehydrogenase deficiency: a historical perspective," Blood, vol.
Rutigliano et al., "Metabolomic profile of glycolysis and the pentose phosphate pathway identifies the central role of glucose-6-phosphate dehydrogenase in clear cell-renal cell carcinoma," Oncotarget, vol.
Glycogen phosphorylase a (GP) activity was determined using an assay kit (Baoman, Shanghai, China) based on the coupled reactions of GP with phosphoglucomutase and glucose-6-phosphate dehydrogenase which resulted in the formation of NADPH.
The other diseases covered by the tests are celiac disease, alpha-1 antitrypsin deficiency, early onset primary dystonia, Factor XI deficiency, Gaucher disease type 1, glucose-6-Phosphate Dehydrogenase deficiency (also known as G6PD), hereditary hemochromatosis, and hereditary thrombophilia.
Kawamoto et al., "A rapid single-step screening method for glucose-6-phosphate dehydrogenase deficiency in field applications," Japanese Journal of Tropical Medicine and Hygiene, vol.
Tang, "Human glucose-6-phosphate dehydrogenase (G6PD) gene transforms NIH 3T3 cells and induces tumors in nude mice," International Journal of Cancer, vol.
Another key enzyme, glucose-6-phosphate dehydrogenase (G6PdH), acts on the alternative pentose monophosphate pathway and is responsible for maintaining appropriate levels of NADPH in the cells (ASAI et al., 2011).
Hyperbilirubinemia and kernicterus in glucose-6-phosphate dehydrogenase deficient infants in Singapore.
Glucose-6-phosphate dehydrogenase deficiency (G6PD deficiency) also known as "favism" is an X-linked recessive genetic condition that predisposes to hemolysis1 and resultant jaundice in response to triggers, such as hypoxia, certain foods, illness and medication2.

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