ciliata decreased the levels of tumor markers: aspartate aminotransferase, alanine aminotransferase, lactate dehydrogenase, alpha-fetoprotein, gamma glutamyl transferase, 5' nucleotidase and glucose-6-phosphate dehydrogenase
. It was concluded that bioactive compounds B.
Severe neonatal hyperbilirubinemia: a potential complication of glucose-6-phosphate dehydrogenase
Beutler, "Glucose-6-phosphate dehydrogenase
deficiency: a historical perspective," Blood, vol.
Rutigliano et al., "Metabolomic profile of glycolysis and the pentose phosphate pathway identifies the central role of glucose-6-phosphate dehydrogenase
in clear cell-renal cell carcinoma," Oncotarget, vol.
Alpha Amylase and Glucose-6-phosphate Dehydrogenase
Glycogen phosphorylase a (GP) activity was determined using an assay kit (Baoman, Shanghai, China) based on the coupled reactions of GP with phosphoglucomutase and glucose-6-phosphate dehydrogenase
which resulted in the formation of NADPH.
The other diseases covered by the tests are celiac disease, alpha-1 antitrypsin deficiency, early onset primary dystonia, Factor XI deficiency, Gaucher disease type 1, glucose-6-Phosphate Dehydrogenase
deficiency (also known as G6PD), hereditary hemochromatosis, and hereditary thrombophilia.
Kawamoto et al., "A rapid single-step screening method for glucose-6-phosphate dehydrogenase
deficiency in field applications," Japanese Journal of Tropical Medicine and Hygiene, vol.
Tang, "Human glucose-6-phosphate dehydrogenase
(G6PD) gene transforms NIH 3T3 cells and induces tumors in nude mice," International Journal of Cancer, vol.
Another key enzyme, glucose-6-phosphate dehydrogenase
(G6PdH), acts on the alternative pentose monophosphate pathway and is responsible for maintaining appropriate levels of NADPH in the cells (ASAI et al., 2011).
Hyperbilirubinemia and kernicterus in glucose-6-phosphate dehydrogenase
deficient infants in Singapore.
deficiency (G6PD deficiency) also known as "favism" is an X-linked recessive genetic condition that predisposes to hemolysis1 and resultant jaundice in response to triggers, such as hypoxia, certain foods, illness and medication2.