Serum biochemistry revealed azotemia (10.3 mmol/L; reference range 3.0-10.0 mmol/L) and
hyperproteinemia (85.3 g/L; reference range 55-75 g/L) with hyperglobulinemia (48.2 g/L; reference range 25-45 g/L) indicating chronicity of infection and possibility due to dehydration.
CHF is associated with interstitial and intravascular volume retention, and patients usually show relative
hyperproteinemia which could influence the quantitative bioactivity of heart failure serum (HFS) as compared to control serum (CS).
Complete blood count results often are helpful in identifying infectious diseases, such as aspergillosis, as many avian patients commonly suffer a heterophilic leukocytosis of 20 X [10.sup.3] cells/[micro]L or more, lymphopenia,
hyperproteinemia, and nonregenerative anemia.
Hyperproteinemia (serum protein >8.0 g/dl) was observed in 13% patients.
What is the possible cause of such extreme
hyperproteinemia in this patient?
In the current study, marked reactive astrogliosis was observed from 42 dpi coinciding with significant pleocytosis and
hyperproteinemia as well as IL-6 increase in the CSF.
Similarly
hyperproteinemia attributable to a polyclonal gammaglobulinaemia is characteristic of chronic abscess.
For example in the presence of hyperlipidemia or
hyperproteinemia, measured serum sodium can also be depressed without concomitant depression of serum osmolarity.
Our finding in
hyperproteinemia (HP) SOME PARAMETERS WERE decreased such as (Vitamin E, LDL and copper) and others were increased such as (GSH, Zinc, Vit.C, uric acid and allantoin) while HDL and transferring saturation were unaffected
This
hyperproteinemia may be attributed to the change in protein metabolism of stressed mice or the increase in the globulin component [20,40].
As observed in rodents and humans, diabetic animals in group M showed hyperglycemia, hypertriglyceridemia and (to a lower extent)
hyperproteinemia that can be considered as results of hyperphagia and also the lack of blood metabolites entering into the tissues.
Other serum index changes included marked hyperchloremia and
hyperproteinemia. Phagocytic activity of hemocytes was significantly depressed (~60%) in treatment lobsters after 14 days and remained so until the end of the experiment.