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Related to hypothyroidism: hyperthyroidism, Levothyroxine


see thyroid glandthyroid gland,
endocrine gland, situated in the neck, that secretes hormones necessary for growth and proper metabolism. It consists of two lobes connected by a narrow segment called the isthmus. The lobes lie on either side of the trachea, the isthmus in front of it.
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decreased functioning of the thyroid gland. Hypothyroidism is a mild form of myxedema. Its basic symptoms are fatigue, slowing down of physical and mental activity, somnolence, sluggishness, decreased memory, sensitivity to cold, puffiness of the face, edematous eyelids, dryness of skin, loss of hair, constipation, and lowered basal metabolism. The treatment for hypothyroidism is hormone therapy.


Condition caused by deficient secretion of the thyroid hormone.
References in periodicals archive ?
4]) in older patients is 25-50 meg/day The TSH level should be rechecked after 6 weeks in patients with overt hypothyroidism and 6-10 weeks in those with subclinical hypothyroidism, with [LT.
Subclinical hypothyroidism is a common disorder with a prevalence of about 7% to 8% in women.
This is thought to be due to the development of an imbalance in the alpha and beta adrenergic receptors with long-term hypothyroidism (3).
uk for |more information about hypothyroidism and other books by Donna and Coralie.
Some newborn babies have congenital hypothyroidism (CH), which is a condition in which thyroid hormone deficiency is present at birth.
Regular hypothyroidism can be diagnosed by a blood test for adequate thyroid hormones but hypothyroidism 2 is caused not by an absence of the thyroxin hormone but by the hormone's inability to enter the cells where it can raise body temperature.
8] It is estimated that the lifetime prevalence of hypothyroidism in DS is 30-50%.
6 Insufficient dietary iodine, which is essential for the production of thyroid hormones, can cause hypothyroidism, but it is rare in developed countries.
Hypothyroidism occurs when the gland produces an insufficient quantity of hormones; when the bodies' cells are unable to use thyroid hormones; or when the gland is surgically removed.
Conclusions: Because of the shift from euthyroidism to spontaneous hypothyroidism, GD patients demanded a strict follow up after ATD therapy.
131]I and hypothyroidism is still unclear, even though Ostroumova et al.