Rao, Synthesis and Mesomorphism of New Chiral Imines
and Copper(II) Complexes, J.
This result is due to the contribution of less reactive imine
groups of PANI component in the cure reaction of epoxy , As a result, when the grafted PANI in the filler increases, the concentration of the imine
groups in the filler increases, and lead to higher [E.sub.a].
Irradiation of azomethine imine
1-4 O solutions in acetonitrile with light of [[lambda].sub.irr] 365 nm results in spectral changes in the characteristic of negative photochromism due to intramolecular photocyclization into diaziridines 1-4 C (Scheme 2), accompanied by a decrease in the intensity of long-wave absorption maxima and the appearance of absorption bands in the short-wave region of the spectrum [10, 30, 31] as shown in Figure 2(a) in the example of compound 1 and in Table 1.
This band is shifted to longer wavelength (lower wavenumber) in the range 1650-1634 [cm.sup.-1] of the complexes and confirms the coordination of the imine
nitrogen to the metal ion.
The reaction of acetaldehyde with most amino acids generates relatively free and stable imines
Hiebl et al., "Unprecedented, low cytotoxicity of spongelike calcium phosphate/poly(ethylene imine
) hydrogel composites," Macromolecular Bioscience, vol.
([d.sub.uv], (1,3) (1,4) (2,2) (2,3) (3,3) (3,4) [d.sub.v]) Number of edges 2n 24n 10n-5 48n-6 13n 8n Table 2: Edge partition of propyl ether imine
dendrimers based on degree of end vertices of each edge.
For diclofenac, several possible reactive intermediates have been postulated, including the 2,5- and 2,4'-quinone imines
and both the parent and 4'-hydroxy-diclofenac acyl-glucuronides.
From chemical structure, we know that the carbonyl group (C=O) in some ketone or aldehyde-compounds is replaced by special functional groups, such as azomethine and/or imine
group, to form series of special Schiff bases which are produced by the reaction of aldehydes or ketones with primary amines in the specific conditions [6, 7].
The main mechanism of acetaminophen-induced hepatotoxicity is an increase in oxidative stress and subsequent saturation of the glucuronidation and sulfation pathways of hepatic elimination, leading to more APAP being metabolized to N-acetyl-[rho]-benzoquinone imine