The T1-weighted MRI images showed high signal intensity BG lesions, with sparing of the
internal capsule. [11] The putamen is almost always involved, with variable additional BG lesions.
MSUD edemas mainly involve the cerebellar white matter, brainstem, globus pallidus,
internal capsule, and thalamus (17) and typically occur in areas that are myelinated in normal full-term neonates (18).
The cranial magnetic resonance imagings (MRIs) of patient 2 revealed abnormal signals in the splenium and genu of the corpus callosum in the first episode and in the bilateral posterior limbs of the
internal capsule and splenium of the corpus callosum in the second episode.
The expression of TLR2 in rat cortex, corpus callosum, and
internal capsule was measured at 1 d, 3 d, and 7 d after DAI.
On MRI, FLAIR sequence showed bilateral and symmetrical hyperintensities in the posterior limb of the
internal capsule, occipital lobe white matter, and splenium of the corpus callosum (Figure 1).
One important point is that in the majority of the cases the insular arteries primarily supply the insular cortex, extreme capsule, and, occasionally, the claustrum and external capsule, but not the putamen, globus pallidus, or
internal capsule, which are vascularized by the lateral lenticulostriate arteries (LLAs), although some thicker branches from M2 may anastomose with the LAAs.
By doing this he confirms the therapeutic effect and avoids any damage to the surrounding
internal capsule. Stereotactic placement of a DBS electrode in these thalamic nuclei, or in the GPi, has a similar effect, with the added benefit of removal if there are any side-effects and the ability to modify the stimulation parameters.
Right postero-lateral thalamic hemorrhage with extension into
internal capsule and lateral ventricle.
This patient suddenly showed a disturbance of consciousness immediately after developing her first lacunar infarct at the posterior limb of the right
internal capsule, and eventually demonstrated severe cognitive disorder.
The overlapping lesions of the brain included the putamen, thalamus,
internal capsule, corona radiata, and primary and premotor cortices (Figure 1: the color represents the frequency of overlap).
Here, we report the case of a 40-year-old male with episodes of paroxysmal non-kinesigenic dystonia (PNKD) as the first manifestation of multiple sclerosis (MS), secondary to an acute demyelinating lesion in the left posterior limb of the
internal capsule detected by magnetic resonance imaging (MRI).
The
internal capsule has also been extensively studied.
