LPL

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Related to lipoprotein lipase: Lipoprotein lipase deficiency

LPL

List Programming Language. LISP-like language with ALGOL-like syntax, for IBM 360. "LPL - LISP Programming Language", F.W. Blair et al, RC 3062, IBM TJWRC, Sep 1970.
References in periodicals archive ?
Genetic study of common variants at the apo E, apo Al, apo Clll, apo B, lipoprotein lipase (LPL) and hepatic lipase (LIPC) genes and coronary artery disease (CAD): variation in LIPC gene associates with clinical outcomes in patients with established CAD.
Prescription omega-3 fatty acids induce a significant elevation of lipoprotein lipase mRNA expression in adipose tissue and post-hepatic lipoprotein lipase, reducing plasma triglycerides, attenuating the post-prandial triglycerides response, and decreasing small dense LDL cholesterol.
Hetrogenous mutations in the Human Lipoprotein gene in patients with Lipoprotein Lipase Deficiency.
These drugs inhibit VLDL secretion, increase lipoprotein lipase activity, and decrease triglyceride levels up to 50%.
The reason Bensadoun's work has a high level of public health interest is that there is now a well-established set of data showing a direct correlation between heart diseases like atherosclerosis, myocardial is chemia, and coronary artery disease and the activity of lipoprotein lipase.
Studies conducted to date have revealed a number of modulator genes, including angiotensinogen, tumor necrosis factor-alpha, glutathione S-transferase P1, lipoprotein lipase, HLA-G, methylenetetrahydrofolate reductase, factor V Leiden mutation, plasminogen activator inhibitor-1, and insulin-like growth factor 11.
The hereditary types of hyperlipoproteinemia are familial lipoprotein lipase deficiency and/or apoprotein C-II deficiency (type I or V), familial hypercholesterolemia (type IIa or IIb), familial dysbetalipoproteinemia type III), familial hypertriglyceridemia (type IV), and combined hypercholesterolemia (type IIa, IIb, and/or IV) (table).
The gene handles the activities of lipoprotein lipase (LPL), an enzyme that helps break down fat particles, known as triglycerides, in the blood.
Lipoprotein lipase is the "gatekeeper" for the fat cells.
Very high levels (greater than 1,000 mg/dl) may be associated with the congenital absence of lipoprotein lipase or apolipoprotein C-2, the latter disorder usually manifesting itself in childhood.

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