Corpus Luteum

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Related to luteal: short luteal phase, Luteal cells

corpus luteum

[′kȯr·pəs ′lüd·ē·əm]
(histology)
The yellow endocrine body formed in the ovary at the site of a ruptured Graafian follicle.
McGraw-Hill Dictionary of Scientific & Technical Terms, 6E, Copyright © 2003 by The McGraw-Hill Companies, Inc.
The following article is from The Great Soviet Encyclopedia (1979). It might be outdated or ideologically biased.

Corpus Luteum

 

in mammals and man, an endocrine gland that develops in the ovary at the site of the graafian follicle after rupture of the follicle wall and discharge of the ovum (ovulation); it consists of altered follicular (so-called lutein) cells. If pregnancy does not follow ovulation, the corpus luteum degenerates within one or two weeks (periodic, or menstrual, corpus luteum); if, however, the ovum is fertilized and pregnancy begins, the corpus luteum grows very rapidly (corpus luteum of pregnancy) and is retained throughout the greater part of pregnancy, secreting the hormone progesterone, which is necessary for the maintenance and development of the pregnancy.

The term “corpus luteum” is sometimes applied to the aggregate of follicular cells formed in the ovary at the site of discharge of the mature ovum in some invertebrates (insects) and in the majority of vertebrates (amphibians, reptiles, and birds).

The Great Soviet Encyclopedia, 3rd Edition (1970-1979). © 2010 The Gale Group, Inc. All rights reserved.
References in periodicals archive ?
Luteal tissue cross-sectional area was measured in cows which were positive for pregnancy on d30 post TAI.
The cause for the sleep disturbances may due to the hormonal changes that occur during the late luteal phase which could have caused a decrease in the RT duration.
The expression patterns of these two steroidogenic enzymes suggest that PRL and LH through its ovarian receptors would indirectly favor luteal P4 production in early and midpregnant vizcachas by both stimulating 3[beta]-HSD and by negatively modulating 20[alpha]-HSD, whereas such modulation would invert at the end of pregnancy allowing the fall of P4 that ultimately elicits parturition in L.
Patients will PMDD have enhanced dorsolateral prefrontal cortex reactivity when anticipating negative stimuli (but not to the actual exposure) during the luteal phase.
However, the P4 AUC in the luteal phase after the 3rd ovulation with estrus, and the AUC for the CL formed after ovulation with estrus between 60 to 80 days after calving (the timing at which farmers generally attempt AI of cows) were significantly greater in the Se-supplemented cows than in the control (Figure 5B: p = 0.04217, 5C: p = 0.00672).
Table-II shows peak and mid luteal E2 in all patients, good responders, and poor responders.
Upon completion of the run-in period, the assessments were conducted during the follicular (9th day of the menstrual cycle) and luteal phases (22nd day of the menstrual cycle), as well as at the beginning of the next menstrual phase [18].
We also strongly recommend that luteal supplementation with hCG should not be used.
Hormone Serum Levels in Our Patients Based on the Menstruation Phases MenstruationS cycle Progesterone (ng/ml) Estradiol (pg/ml) Mensturation phase 25.3 [+ or -] 2.83 42.7 [+ or -] 4.28 Ovulation time 78.47 [+ or -] 3.31 149.36 [+ or -] 16.92 Luteal phase 121.44 [+ or -] 11.56 175.34 [+ or -] 13.71 MenstruationS cycle P Value P ValueinACL Laxity (a)(Lachman) Mensturation phase 0.67 0.67 Ovulation time 0.43 0.83 Luteal phase 0.49 0.7 (a) There are no laxity.
If the patient is in the early luteal phase (progesterone level >3 ng/[micro]L), ovarian stimulation is started without GnRH antagonist.