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Inflammation of lymphatic vessels.



lymphangiitis, inflammation of the lymphatic vessels.

Lymphangitis may develop with inflammation of the skin and mucosa if the infection spreads, with the lymph flow, toward the lymph nodes. The causative agents of the process, including streptococci, staphylococci, and colon bacilli, penetrate from intertissular fissures of the inflamed region first to the efferent surface lymph vessels and later to deeper-lying ones. The entire wall of the vessel is affected. Fibrin clots fall into the lumen, interrupting the flow of lymph (this has significance in circumscribing the inflammation).

Lymphangitis is manifested by narrow red stripes on the skin; in some forms, induration and soreness develop in their vicinity. Simultaneously, the body temperature rises and chills develop. The patient experiences general malaise. Edema and tenderness are observed with lymphangitis of the deep vessels. Chronic lymphangitis is characterized by occlusion of the lymphatic ducts and resultant edema.

Lymphangitis is treated by eliminating the primary focus, resting the affected part of the body, and administering physiotherapeutic procedures, compresses, and antibiotics. With chronic lymphangitis, recommended treatments are physiotherapy, pelotherapy, and X-ray therapy. The condition may be prevented by the timely treatment of inflammatory, traumatic, and other foci.


References in periodicals archive ?
Pulmonary involvement in ECD is characterized microscopically by interstitial accumulations of non-Langerhanstype histiocytes in a lymphangitic distribution pattern (Figure 6, B through D), typically with an abrupt, smooth interface with the adjacent uninvolved alveolar parenchyma.
Lymphangitic carcinomatosis may cause either beaded or smooth septal thickening.
CT findings in lymphangitic carcinomatosis of the lung: correlation with histologic findings and pulmonary function tests.
101,104-107) Therefore, rigorous exclusion of lymphoma is required when lymphoid infiltrates are expansile or tumefactive and track along lymphangitic routes.
The bronchocentric cases may be accompanied by nonnecrotizing granulomas in a lymphangitic distribution.
Biopsy samples generally showed a plasma cell-rich lymphohistiocytic infiltrate with fibrosis in a lymphangitic distribution, increased numbers of IgG4-positive plasma cells on immunohistochemistry (range, 20-231 in 3 high-power fields), an increased IgG4 to IgG plasma cell ratio (range, 10%-72%), and involvement of both arteries and veins by intimal endothelial inflammation (Figure 1, a through g).
Focal and limited lymphangitic spread of lymphocytes and plasma cells around the bronchovascular bundles and interlobular septa may be seen.
83) Exclusion of other diseases in the differential diagnosis appears to be the primary role (eg, infection, sarcoidosis, hypersensitivity pneumonitis, lymphangitic tumor).
Careful examination of the lungs revealed no evidence of lymphangitic metastasis or involvement of the lung parenchyma.
4 patients, the ill-defined nodules were distinctly lymphangitic in distribution, involving broncho-vascular bundles, interlobular septa, and visceral pleura (Figure 1).