mitogenesis


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mitogenesis

[‚mīd·ə′jen·ə·səs]
(cell and molecular biology)
Induction of mitosis.
Formation as a result of mitosis.
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References in periodicals archive ?
Nowadays, a plethora of research studies has confirmed that mitogenesis arises within an inflammatory microenvironment [88], while chronic, low-grade inflammation accompanies the disease course.
These and the abundant cell infiltrate, mitogenesis, and deposition of new ECM residents comprise the most relevant events when biomaterials are employed (Badylak, 2007).
VEGF is a critical endogenous proangiogenic factor that binds and activates VEGF receptor to induce angiogenesis, mitogenesis, and Chemotaxis in endothelial cells.
This has a number of intracellular effects, which ultimately promote cell survival and mitogenesis.
Endothelial dysfunction is another major factor resulting in vasoconstriction, thrombosis, and mitogenesis through imbalanced production of vasoconstrictors and vasodilators, prothrombotic and antithrombotic mediators, and activators and inhibitors of smooth muscle cell growth and migration.
Reproductive events involving leptin are crucial for adequate functional development of the placenta, including regulation of nutrient transport, placental angiogenesis, trophoblast mitogenesis, and immunomodulation (Henson and Castracane 2000).
Tavernarakis, "Coupling mitogenesis and mitophagy for longevity," Autophagy, vol.
Hung et al., "Role of the Janus kinase (JAK)/signal transducters and activators of transcription (STAT) cascade in advanced glycation end-product-induced cellular mitogenesis in NRK-49F cells," Biochemical Journal, vol.
Another possible mechanism of Cd carcinogenicity assumed by the same authors [24] is the ability of Cd to induce mitogenesis in pancreatic cells as reflected by the increased metallothionein synthesis to which Cd is highly bound in the pancreas of rats.
The NFkB pathways have also been implicated in NTS-induced inflammation and mitogenesis in colonocytes [35].
Mitogenesis of vascular smooth muscle cell stimulated by platelet-derived growth factor-bb is inhibited by blocking of intracellular signaling by epigallocatechin-3-O-gallate.
G-protein-coupled receptor genes as protooncogenes: Constitutively activating mutation of the alpha 1B-adrenergic receptor enhances mitogenesis and tumorigenicity.

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