myenteric


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Related to myenteric: myenteric plexus, myenteric reflex

myenteric

[¦mī·ən¦ter·ik]
(histology)
Of or pertaining to the muscular coat of the intestine.
References in periodicals archive ?
Overall number of ganglion cells in the myenteric plexus between longitudinal and circular muscle layer of adult colon was 16.
13,14) In the literature, a study reported that the rate of achalasia was elevated among patients with BN compared to the general population, (7) and there are some case reports about BN patients developing achalasia because of myenteric plexus damage.
Histologically, Hirschsprung disease is characterised by the absence of ganglionic cells in the myenteric and submucous plexuses and the presence of hypertrophied non-myelinated nerve trunks in the space normally occupied by the ganglionic cells.
In the early stages of PD, decreased GI motility has been associated with neuronal loss in the myenteric and submucosal plexi and inclusions of Lewy bodies in the dorsal motor nucleus of the vagus, underscoring their potential role in slowing down intestinal peristalsis [7, 28, 32].
Diffuse ganglioneuromatosis refers to masslike intramural or transmural expansions of the myenteric plexus and nerve tracts by neural, glial, and stromal elements and are associated with neurofibromatosis 1 and multiple endocrine neoplasia type IIb.
Effects of oxaliplatin on mouse myenteric neurons and colonic motility.
Collectively, they represent the myenteric (Auerbach's) plexus, which provides innervations of the muscle layers.
It is documented that peripheral injection of CRF or urocortin stimulates colonic transit, motility through Fos gene expression in myenteric neurons, and defecation as a result of [CRF.
Ultrastructurally they reveal features suggestive of their myenteric plexus origin such as neuron-like cells with cytoplasmic processes, intermediate filaments, bulbous synapse-like, dense core neurosecretory granules.
Peripheral CRF activates myenteric neurons in the proximal colon through CRF(1) receptor in conscious rats.
In accordance with WILDER-SMITH & BETTIGA (1997), the interference of tramadol with GI motility is related to the direct influence of the drug over the myenteric plexus by the central modulation via opioid or by monoaminergic mechanisms.
Ogren, "Effects of alaproclate, potassium channel blockers, and lidocaine on the release of 3-Hacetylcholine from the guinea-pig ileum myenteric plexus," Pharmacology & Toxicology, vol.