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Elevation of the leukocyte count to values above the normal limit.
McGraw-Hill Dictionary of Scientific & Technical Terms, 6E, Copyright © 2003 by The McGraw-Hill Companies, Inc.
The following article is from The Great Soviet Encyclopedia (1979). It might be outdated or ideologically biased.



an increase in the absolute number of leukocytes in the peripheral blood.

Physiological leukocytosis occurs after eating and physical exertion. Symptomatic pathological leukocytosis develops with suppurative and inflammatory processes and with a number of infectious diseases, whether produced by specific causative agents of infection or as a result of the bone-marrow reaction to the breakdown of tissues caused by toxic agents or circulatory disorders (for example, myocardial infarction). Transient leukocytosis may result from the release of leukocytes into the blood from bone marrow or other tissues (for example, in stress). In all cases, the leukocytosis is reactive and disappears when its cause disappears. Leukocytosis may be produced by a malignant proliferation of hematopoietic tissue, as in leukemia.


Kassirskii, I. A., and G. A. Alekseev. Klinicheskaia gematologiia, 4th ed. Moscow, 1970.
The Great Soviet Encyclopedia, 3rd Edition (1970-1979). © 2010 The Gale Group, Inc. All rights reserved.
References in periodicals archive ?
In the current case, a few days before admission, the patient's clinical and laboratory findings were nonspecific (pruritic erythematous-edematous plaques, neutrophilic leukocytosis, and increased inflammatory markers) and suggestive of a pathological condition, which led the clinician to an incorrect diagnosis (urticaria and mild fever).
The laboratory findings presented significantly higher percentage of leukocytosis for age, neutrophilic leukocytosis for age, and positive CRP in E.
injected and gavaged mice within treatment groups suggesting that both routes of ethanol administration induce an equal neutrophilic leukocytosis after burn injury.
Blood investigations showed a neutrophilic leukocytosis with deranged renal and liver function, and prolonged INR.
Amongst the laboratory parameters neutrophilic leukocytosis was seen in 28(58.3%) cases.
Clinical laboratory evaluation showed the following: neutrophilic leukocytosis, hemoglobin 10.1 g/dL, hematocrit 30.3%, thrombocytopenia (40 x [10.sup.6] platelets/L [reference range 140-440 x [10.sup.6] platelets/L]), elevated serum alkaline phosphatase (290 U/L [reference range 35-104 U/L]), elevated lactic dehydrogenase concentration (560 U/L [reference range 100-190 U/L]), and elevated antistreptolysin O and rheumatoid factor titers.
Marked neutrophilic leukocytosis with shift to left of reticulocytes (immature/band cells) was indicative of severe bacterial infection to uterus, which might be due to stimulation of bone marrow to release more number of immature or band cells into peripheral circulation in an attempt to overcome infection (Mojzisova et al., (2000) (Fig.
Laboratory investigations showed mild neutrophilic leukocytosis with total white cell count (11.4 x [10.sup.9] per liter).
The combination of urticaria rash, fever, arthralgia, polyserositis, and neutrophilic leukocytosis in the absence of specific autoantibodies suggested a differential diagnosis of adult-onset Still's disease (AOSD).
Male patient, 75 years old, with history of a right inguinal mass that progressively grew over several years, went to emergency room of Mantua General Hospital for right inguinal pain, associated with fever and neutrophilic leukocytosis, without signs of intestinal obstruction.