Oliguria

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oliguria

[′äl·ə‚gyu̇r·ē·ə]
(medicine)
Diminished excretion of urine.

Oliguria

 

a decrease in the daily quantity of excreted urine from the normal 1,500 ml to 500 ml. Oliguria results from reduced filtration or increased reabsorption in the kidneys. Physiological oliguria arises with dehydration, which can result from water deprivation, excessive perspiration, vomiting, or diarrhea. It may also accompany blood loss; extensive burns and traumas; a drop in arterial pressure, as in shock and collapse; or increased production of the hormones aldosterone or vasopressin. Oliguria is frequently a symptom of kidney disease, for example, nephritis.

References in periodicals archive ?
In our study, out of 30 cases of renal failure, 24 cases (80%) were oliguric with mortality of 41.
Patients of ARF were further divided into two groups, namely oliguric and nonoliguric renal failure based on daily urine output of <400 ml (or <0.
Increasing plasma potassium and sodium, and water retention in the oliguric phase of AKI cause disruption to fluid and electrolyte balance throughout the body.
Both studies found high doses of furosemide could induce a high urine output and convert oliguric renal failure to non-oliguric renal failure without reducing the need for dialysis and/or decreasing mortality.
15) found a significantly higher mortality in patients with oliguric leptospirosis than in non-oliguric forms.
However, even with the wealth of information available from observed parameters, the ICU staff commonly deal with difficult questions such as, should an oliguric patient be treated with fluids or with diuretics, and are they suffering from hypovolaemia or fluid overload?
This stage is followed by an oliguric stage as kidney function improves.
HUS is a TMA that mainly affects the kidney and may cause oliguric or anuric renal failure.
As with saline hydration, sodium bicarbonate hydration should be used with caution in patients with hypertension, congestive heart failure, or oliguric renal failure, due to the risk of sodium retention and volume overload.
In the third, oliguric phase, hypertension may develop due to diminished urine formation.
In 1990, Davenport and Will examined 8 consecutive adult patients suffering from fulminant hepatic failure and oliguric renal failure after paracetamol (acetaminophen) self-poisoning.