Periodontitis


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periodontitis

[¦per·ē·ō‚dän′tīd·əs]
(medicine)
Inflammation of the periodontium.

Periodontitis

 

an acute or chronic inflammation of the periodontium and adjacent tissues. Periodontitis is usually a result of dental caries and arises as infection spreads from the root canal through the apical foramen to the apex of the root. It may also develop from frequently recurring injury to the tooth; this kind of injury can result from such occupational habits as biting thread or grasping metal nails with the teeth and from any habitual biting of hard objects, for example, pencils or the mouthpieces of pipes.

Acute periodontitis is manifested by sharp pain in the region of the tooth that intensifies when the inflamed area is touched. Swelling often occurs in the gums, lips, or cheeks, and the affected tooth becomes loose. The enlarged submaxillary lymph nodes become sensitive, and fever occasionally arises. The process may sometimes be complicated by osteomyelitis of the jaw, purulent inflammation of the soft tissues of the face and neck, and acute sepsis. Symptoms of chronic periodontitis usually include discomfort while chewing, halitosis, and sometimes fistulas on the gums and facial skin. Chronic periodontitis can lead to the formation of a cyst of the jaw, and both forms of periodontitis can sensitize the body to streptococci.

Treatment of periodontitis is usually conservative, consisting of filling the root canals. Often the tooth is removed. Abscesses are lanced, and antibiotics are used to treat pronounced general symptoms.

REFERENCES

Groshikov, M. I. Periodontit. Moscow, 1964.
Marchenko, A. I. “Bolezni periodonta.” In Rukovodstvo po terapevticheskoi stomatologii. Moscow, 1967.
Ovrutskii, G. D., F. G. Gasimov, and S. V. Makarov. Bolezni zubov. Kazan, 1967.
Rybakov, A. I., and V. S. Ivanov. Klinika terapevticheskoi stomatologii. Moscow, 1973.

G. D. OVRUTSKII

References in periodicals archive ?
It is an acute phase reactant protein and its levels are elevated up to 20% in acute inflammation.7 Recent studies have found DBP to be widely distributed in periodontal tissues where it is highly expressed, thereby speculating its important role in local immune defense.8 There are few studies reporting the role of 1,25(OH)2D and DBP in periodontitis. In this study, we explored the relationship between 1,25(OH)2D and DBP in patients with periodontitis and healthy controls.
El proposito de esta investigacion fue evaluar los niveles de las interleucinas IL-1[alfa], IL-1[beta], TNF-[alfa] y los parametros clinicos en pacientes diagnosticados con periodontitis, antes y despues de la TPNQ.
This study investigated the effects of AsIV on inflammation and immunity in rats with experimental periodontitis. The results indicate that AsIV can mitigate periodontitis in rats.
The researchers found that in the presence of periodontitis, mean systolic BP was about 2.3 to 3.0 mm Hg higher among treated adults with hypertension.
[8] Hence, the present study was aimed to investigate the MPV value and its relation with periodontitis and change in value of MPV after non-surgical periodontal therapy.
It is characterized by palmoplantar hyperkeratosis and aggressive periodontitis, has a worldwide prevalence of 1-4 cases per million in the general population and is often related with consanguinity in 20-40% of patients with PLS.1,2 It usually has its onset between the ages of 1 and 4 affecting males and females equally.3 Cutaneous manifestations begin with erythema which progress to hyperkeratosis of soles and palms within 6 months.2 Palmoplantar keratosis, varying from mild psoriasiform scaly skin to overt hyperkeratosis is characteristic.
Overall, the resulting new classification is very different from the prior one as the periodontitis category is based on the oncology system of "Staging" and "Grading" of disease.
Diferentes variables ambientales, sociodemograficas y biologicas se han evaluado como posibles factores de riesgo de periodontitis (3).
As an increasing number of patients receive implants to replace missing teeth lost due to periodontitis, the question arises as to whether a history of periodontitis may increase the risk of peri-implant disease (e.g.
Periodontitis, also called gum disease or periodontal disease, is known as a "silent" disease because it is not always painful.
Purpose: The purpose of this study was to determine the expression of recently discovered pro--and anti-inflammatory mediators: TREM-1, TREM-2, RAGE, HMGB-1, and their soluble forms: sTREM-1, sTREM-2, and sRAGE in gingival crevicular fluid (GCF) during gingivitis and periodontitis to discover an appropriate target for future molecular interventions.