The Porta hepatis is centrally located in the middle third of the visceral surface; it has an "L" shape rotated ninety degrees to the left; it presents a transverse part, another anterior part, and they form a 90-degree angle of anterior medial sinus; the hepatic portal vein
is divided here into right (A12.3.12.002) and left (A12.3.12.005) branches; the hepatic artery proper is divided into right (A12.3.12.030) and left (A12.3.12.035) branches; the common hepatic duct is formed by the union of the right hepatic (A05.8.01.062) and the left hepatic (A05.8.01.065); these branches form the first vascular division; then, on the edge of this parenchymal channel, these two "primary branches" emit seven terminal secondary branches, forming seven vascular pedicles for seven portal segments.
(8) published the first study on post liver transplantation portal vein
angioplasty and stent insertion, percutaneous transhepatic balloon angioplasty and stent insertion have been commonly used and are considered safe methods to treat PVS after liver transplantation (9-11).
Preoperative patient data included age, gender, etiology of cirrhosis, hypertension, diabetes mellitus, Child-Pugh classification, the longitudinal spleen diameter, the main portal vein
diameter, spleneolus, hemoglobin levels, the PLT count, prothrombin time, and levels of total bilirubin, plasma albumin, alanine aminotransferase, blood urea nitrogen, creatinine, laminin, precollagen III, collagen IV, and hyaluronidase.
Bolognesi et al showed that portal pressure could be predicted by using a formula using the splenic PI and the portal vein
flow22 splenic artery pulsatility index, thus, offers a non-invasive method of assessing the splenoportal haemodynamics and helps in predicting the status of the hepatic fibrosis and the resulting changes in the portal system due to chronic liver disease22.
Axial images demonstrated formation of the portal vein
by confluence of the splenic and superior mesenteric veins at the neck of pancreas, but instead of coursing into the liver through the porta hepatis, this vein formed an end-to-side anastomosis with the extra hepatic IVC (Figure 1).
The right portal vein
was transected with a vascular staple; the right hepatic artery, vein, and right hepatic canal were saved (Figure 2, 3), and the parenchymal dissection of the right liver lobe was performed with an ultrasonic dissector and isolated with a sterile plastic bag (Figure 4).
Dynamic computed tomography scans showed fusiform aneurysmal enlargement in the proximal segment of the intrahepatic right portal vein
(Figure 2a, 2b).
Cavernous transformation of the portal vein
(CTPV) among adults is quite rare, with an incidence of 15.6% among extrahepatic portal vein
obstruction (EHPVO), including one-third of thrombotic portal veins
, and accordingly are thought to be secondary to EHPVO triggered by inflammation, adjacent tissue fibrosis, tumor invasion, pancreatitis, and clotting disorders.,, CTPV is characterized by a series of tortuous collateral portal veins
at the liver hilum, with portal vein
occlusion at the level of the spinoportal conjunction. In this case, it was more likely caused by chronic cholecystitis based on the original normal portal vein
Increased NO secretion is one of the main responsible mediators that occurred from splanchnic vein hyperemia and vasodilatation of the portal vein
The tumor factors include the maximum tumor diameter (MTD), number of tumors, presence of macroscopic (clinically evident) portal vein
thrombosis (PVT), and blood levels of alpha-fetoprotein (AFP).
Portal hypertension (PH) refers to a group of clinical syndromes characterized by abnormal changes of hemodynamics in portal vein
The umbilical vein divides into two structures at the liver (ductus venosus and a branch that communicates with the hepatic portal vein