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Both foci of adenosis and sclerosing adenosis were present in 2 cases.
Pathology yielded 7 classic fibroadenomas, 2 juvenile fibroadenomas, 2 tubular adenomas, 1 nodular sclerosing adenosis (Fig.
Lesions were classified as fibrocystic change with or without epithelial hyperplasia if the lesion had apocrine metaplasia, nodular adenosis, sclerosing adenosis, papillomatosis, and usual ductal hyperplasia.
Special techniques for example morphometry, DNA ploidy and immunohistochemical studies against various antigens are used to differentiate premalignant lesions like fibrocystic disease with epitheliosis, atypical ductal hyperplasia, atypical lobular hyperplasia, sclerosing adenosis and carcinoma in situ.8
Of the two sclerosing adenosis without atypia, irregular and smooth margin as well as irregular and round shape was having equal distribution.
Therefore, core biopsy was performed and revealed sclerosing adenosis. Diagnosis of sclerosing adenosis can be difficult with mammogram, ultrasound, and MRI, and tissue diagnosis may be required for definitive diagnosis.
4a and 4b), sclerosing adenosis or a radial scar.11 We conclude that all spontaneous nipple dis- charge regardless of their color be referred to a breast surgeon for triple assessment.
The most common benign lesion encountered was fibrocystic change, followed by sclerosing adenosis. We found fibroadenoma, classically a condition encountered in younger individuals, to be more common among older patients.
(2-3) Complex fibroadenomas contain other proliferative changes such as sclerosing adenosis, duct epithelial hyperplasia, and epithelial calcification and are associated with slightly increased risk of cancer.
The false positive results occur because of some benign diseases with hypercellularity like cellular fibroadenoma, proliferative fibrocystic disease, phyllodes tumor, lactational changes, sclerosing adenosis, and so forth, and in lesions with atypia like postradiation, fat necrosis, and radial scar [9].
Benign lesions included fibrocystic disease, sclerosing adenosis, fibroadenoma, ductal ectasia and lipoid necrosis.
Epithelial proliferation was frequently accompanied by intraductal papillomatosis, components of fibrocystic disease (apocrine metaplasia and hyperplasia, fibrosis, ductal ectasia), fibroadenoma, adenosis, sclerosing adenosis and chronic mastitis.