fibrosing adenomatosis

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fibrosing adenomatosis

[fī′brōs·iŋ ‚ad·ən‚ō·mə′tō·səs]
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Lesions were classified as fibrocystic change with or without epithelial hyperplasia if the lesion had apocrine metaplasia, nodular adenosis, sclerosing adenosis, papillomatosis, and usual ductal hyperplasia.
Special techniques for example morphometry, DNA ploidy and immunohistochemical studies against various antigens are used to differentiate premalignant lesions like fibrocystic disease with epitheliosis, atypical ductal hyperplasia, atypical lobular hyperplasia, sclerosing adenosis and carcinoma in situ.
Of the two sclerosing adenosis without atypia, irregular and smooth margin as well as irregular and round shape was having equal distribution.
Diagnosis of sclerosing adenosis can be difficult with mammogram, ultrasound, and MRI, and tissue diagnosis may be required for definitive diagnosis.
The most common benign lesion encountered was fibrocystic change, followed by sclerosing adenosis.
2-3) Complex fibroadenomas contain other proliferative changes such as sclerosing adenosis, duct epithelial hyperplasia, and epithelial calcification and are associated with slightly increased risk of cancer.
Benign lesions included fibrocystic disease, sclerosing adenosis, fibroadenoma, ductal ectasia and lipoid necrosis.
Complex fibroadenoma refers to fibroadenomas with foci of cysts, sclerosing adenosis, epithelial calcifications, and papillary apocrine metaplasia, and has a higher future risk of malignancy.
In contrast, in cases of sclerosing adenosis, a benign breast lesion, ATM is expressed in both the epithelial and myoepithelial cells.
Epithelial proliferation was frequently accompanied by intraductal papillomatosis, components of fibrocystic disease (apocrine metaplasia and hyperplasia, fibrosis, ductal ectasia), fibroadenoma, adenosis, sclerosing adenosis and chronic mastitis.
5) Diagnostic results can be false positive due to misinterpretation of lobular cancerization and sclerosing adenosis.
Microscopically there was a non-encapsulated lesion with a combination of stroma and cells in the terminal duct lobular units and there was superimposed sclerosing adenosis indicative of a hamartoma.