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see arrhythmiaarrhythmia
, disturbance in the rate or rhythm of the heartbeat. Various arrhythmias can be symptoms of serious heart disorders; however, they are usually of no medical significance except in the presence of additional symptoms.
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an increase in the frequency of cardiac contractions. In some cases it is not perceived subjectively; in others, it is accompanied by palpitations.

A distinction is made between sinus tachycardia, or the accelerated generation of impulses in the sinus node of the heart, and paroxysmal tachycardia. Sinus tachycardia, manifested by contractions generally ranging between 90 and 120 per minute, may be caused by such physiological factors as increased environmental temperature, physical and mental tension, or the ingestion of food. Sinus tachycardia may also be caused by such pathological states as fever, anemia, diffuse toxic goiter, heart failure, and neurasthenia. Nervous and humoral influences on the heart, such as adrenalin and thyroxine, are important contributing factors in the genesis of sinus tachycardia. The accelerated rhythm of cardiac contractions may have an unfavorable effect on metabolism in the myocardium and on blood circulation. When tachycardia is a symptom of disease, the underlying disease is treated.


Excessive rapidity of the heart's action.
References in periodicals archive ?
Globally the incidence of SCD is expected to rise sharply as the prevalence of coronary artery disease and heart failure continue to increase.18 Furthermore ventricular fibrillation (VF) and sustained ventricular tachycardia (VT) are also major causes of cardiac mortality and both of these arrhythmias occur as a result of a complex interplay of abnormal substrates myocardial vulnerability imbalance of autonomic regulation and so on.
The provocation of serious ventricular arrhythmias by flecainide has been described.[11-13] The incidence approaches 20% in patients with severe ventricular dysfunction and a previous history of sustained ventricular tachycardia who are taking more than 400 mg of the drug daily; moreover, in about 10%, the proarrhythmic events were fatal.[11,13] Patients with less severe dysrhythmias (chronic PVCs and nonsustained ventricular tachycardia) have had a 3% rate; 0.1% were fatal.[11] First degree, second degree, and, third degree A-V block have also occurred with flecainide.
The patient presented an episode of sustained ventricular tachycardia, with its origin in the right ventricle or in the interventricular septum, with a heart rate of 200/min, having syncope as the main clinical manifestation; nevertheless, non-sustained ventricular tachycardia originating in the left ventricle was identified on the 24 hour Holter monitoring [1], with RR' morphology in V1, R> R' 100/min, but completely asymptomatic.
Effective: Absence of sustained ventricular tachycardia, the incidence of non-sustained ventricular tachycardia was decreased by over 90%, and PVCs were decreased by over 75%.
The electrocardiogram showed sustained ventricular tachycardia with very fast heart rate of approximately 300 bpm associated with a right bundle branch block pattern (Figure 1(a)).
Sustained ventricular tachycardia and diffuse right ventricular/left ventricular abnormalities are features of phase II disease.
However, the patient suddenly complained of palpitation and sustained ventricular tachycardia thirty minutes after the procedure.
In the 4th hour of his arrival, ECG showed sustained ventricular tachycardia with pulse that responded to medical therapy (Fig.
The expanded ICD coverage includes two more categories of patients-those with coronary artery disease with prior myocardial infarction, left ventricular ejection fraction less than or equal to 35%, and inducible, sustained ventricular tachycardia or ventricular fibrillation at electro-physiological study; and those with a prior myocardial infarction and left ventricular ejection fraction less than or equal to 30% and a QRS duration of greater than 120 milliseconds.