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see arrhythmiaarrhythmia
, disturbance in the rate or rhythm of the heartbeat. Various arrhythmias can be symptoms of serious heart disorders; however, they are usually of no medical significance except in the presence of additional symptoms.
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an increase in the frequency of cardiac contractions. In some cases it is not perceived subjectively; in others, it is accompanied by palpitations.

A distinction is made between sinus tachycardia, or the accelerated generation of impulses in the sinus node of the heart, and paroxysmal tachycardia. Sinus tachycardia, manifested by contractions generally ranging between 90 and 120 per minute, may be caused by such physiological factors as increased environmental temperature, physical and mental tension, or the ingestion of food. Sinus tachycardia may also be caused by such pathological states as fever, anemia, diffuse toxic goiter, heart failure, and neurasthenia. Nervous and humoral influences on the heart, such as adrenalin and thyroxine, are important contributing factors in the genesis of sinus tachycardia. The accelerated rhythm of cardiac contractions may have an unfavorable effect on metabolism in the myocardium and on blood circulation. When tachycardia is a symptom of disease, the underlying disease is treated.


Excessive rapidity of the heart's action.
References in periodicals archive ?
Subjects receiving misoprostol were twice as likely to experience tachysystole.
Inappropriate use of oxytocin is common, including the improper management of oxytocin infusion in the setting of uterine tachysystole (defined as the presence of >5 contractions over a 10-minute period averaged over 30 minutes) and/or an abnormal FHR tracing.
The maternal side effects observed were tachysystole, hyperstimulation, vomiting, diarrhoea, fever and PPH.
As expected, tachysystole (five or more contractions in 10 minutes over a 20-minute period) was more common with misoprostol (20.
When clear definitions of abnormal heart-rate patterns and tachysystole are addressed through Specific interventions, according to this prospective, nonrandomized study of 14,398 women undergoing induction of labor with oxytocin.
Hyperstimulation was defined as tachysystole (at least 6 contractions in 10 minutes) or prolonged uterine contractions > 2 minutes accompanied by abnormal fetal heart rate tracing.
Misoprostol was associated with more tachysystole (16% for oral and 20% for intravaginal administration) than was dinoprostone/oxytocin (2%), but rates of hyperstimulation were similar: 4%, 7%, and 2%, respectively.
Although incidence of tachysystole and hyperstimulation were more in the group induced by Misoprostol, incidence of PPH was considerably less than that in the group induced by Dinoprostone.
5) Oral terbutaline should be avoided entirely in the prevention and treatment of preterm labor, However, the use of terbutaline for the management of acute tachysystole with an abnormal fetal heart-rate (FHR) pattern remains a reasonable course of treatment.
Depending on the clinical situation, efforts to resolve the abnormal pattern include (but are not limited to) provision of maternal oxygen, change in maternal position, discontinuation of labor stimulation, treatment of maternal hypotension, and treatment of tachysystole with FHR changes.
2] group 1% cases had tachysystole, 2% cases had hypertonus and 3% cases had discomfort at the time of insertion.
Induction included cervical ripening with misoprostol followed by oxytocin, resulting in uterine tachysystole and an abnormal fetal heart-rate pattern.