thyrotoxicosis


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Related to thyrotoxicosis: hyperthyroidism, Graves disease

thyrotoxicosis

[¦thī·rō¦täk·sə′kō·səs]
(medicine)
McGraw-Hill Dictionary of Scientific & Technical Terms, 6E, Copyright © 2003 by The McGraw-Hill Companies, Inc.
References in periodicals archive ?
When a patient presents with psychosis as a result of thyrotoxicosis, treatment focuses on improving the thyrotoxicosis through anti-thyroid medications and beta blockers (Table 33).
Furthermore, the initial presentation of thyrotoxicosis signs, as well as imaging and laboratory findings deviated the attention toward to interpret observations as thyroiditis [induced by trauma (FNAB)] with associated swelling of goiter, causing obstructive and bulbar symptoms.
Thyrotoxicosis is characterized by excess circulating thyroid hormones, irrespective of the source (1).
To study the Clinical pattern of presentation of patients with thyrotoxicosis, Indication for surgery in thyrotoxicosis patients and to assess the functional results and quality of life following the surgical treatment
This was a case of thyrotoxicosis. He laid emphasis on rational use of investigations.
In the index case thyrotoxicosis induced liver damage settled after he became euthyroid and liver enzymes normalized as well.
One study18 suggested that Asian pregnant women are more prone to developing gestational thyrotoxicosis, which is a crucial finding also corroborated in our study (8 cases, 2.08% of overt hyperthyroidism).
Hall mark features may be present according to etiology such as exophthalmos in Graves' disease, which is autoimmune and the most common cause of thyrotoxicosis. It's common in whites and Asians.
Low TSH and FT4 are associated with secondary (central) hypothyroidism, non-thyroidal illness (undetectable concentrations not typical), or triiodothyronine (T3) thyrotoxicosis (1).
With the significant increases in thyroid hormones reported in the active intervention, it is important to monitor for ongoing safety and any risk of thyrotoxicosis. Further long-term studies with larger and diverse study populations are required, as well as more studies assessing the clinical outcomes of normalising thyroid indices to better understand optimal treatment strategies.
Thyrotoxicosis (overactive thyroid) which tends to run in families.
Thyroid work-up revealed suppressed TSH at <0.005 (ref: 0.45-5.33) uIU/ml, elevated free T4 at 5.36 (ref: 0.58 -1.64) ng/dl, free T3 of 28.31 (ref: 2.2-4.10) pg/ml along with elevated anti-thyroglobulin antibodies at 12 (ref: [less than or equal to] 4) IU/ml, thyroid peroxidase antibodies at 3841 (ref: [less than or equal to] 8) IU/ml, and thyroid-stimulating immunoglobulin >500% ([less than or equal to] 122%), suggesting Graves' thyrotoxicosis. Thyroid ultrasound showed significantly enlarged, mildly heterogeneous lobes without discrete nodules.