In 1950, after a decade of 11 other unpublished New Zealand cases of tetanus with two recoveries, and another series treated by basal sedation with 50% surviving, DW Beaven (50) and his Christchurch team supplied a six-year-old boy with anaesthetic doses of tribromoethanol
with 350 mg tubocurarine (d-TC) over [greater than or equal to] 12 days, to control his tetanus spasms and severe opisthotonus.
The hamsters were anesthetized with 2.5% tribromoethanol
(10 mL/kg, ip) on days 4 and 10 for evaluation of the salivation rate (22).
After the third ECG recording, at the 6th month of observation, the animals were reanesthetized with tribromoethanol
(250 mg/kg, i.p.), and a pair of stainless steel electrodes were implanted inside the subcutaneous tissue to collect chronic recordings of conventional bipolar limb ECG lead II.
Induction of occlusal alteration.The animals were anaesthetized with tribromoethanol
(0.25g/kg body weight) and submitted to extraction of the upper molars at the left hemiarch, with animals receiving the antibiotic Pentabiotic (24000 IU) as preoperative prophylaxis.
At the end of the session, they were anesthetized with tribromoethanol
(250 mg/kg, ip) and placed in supine position on a flat surface for the insertion of four electrodes via hypodermic needles (two in the head and two in the tail).
The mice were anesthetized with 2.5% tribromoethanol
and restrained on a small board.