Can, "Protective effect of royal jelly in 2,4,6 trinitrobenzene
sulfonic acid-induced colitis in rats," Iranian Journal of Basic Medical Sciences, vol.
Manning, "Use of trinitrobenzene
as a nitrogen source by Pseudomonas vesicularis isolated from soil," Current Microbiology, vol.
Alarcon de la Lastra, "Effects of dosmalfate, a new cytoprotective agent, on acute and chronic trinitrobenzene
sulphonic acid-induced colitis in rats," European Journal of Pharmacology, vol.
Xia, "Curcumin regulated shift from Th1 to Th2 in trinitrobenzene
sulphonic acid-induced chronic colitis," Acta Pharmacologica Sinica, vol.
Qiao, "Superoxide dismutase recombinant Lactobacillus fermentum ameliorates intestinal oxidative stress through inhibiting NF-kappaB activation in a trinitrobenzene
sulphonic acidinduced colitis mouse model," Journal of Applied Microbiology, vol.
In acute models like trinitrobenzene
sulphonic acid (TNBS) induced colitis also, the activity of sesame oil has been explored  but not the active constituent sesamol.
Support for this concept comes from animal studies showing that inhibition of 5-LO is also efficacious in blocking chronic colitis resulting from administration of trinitrobenzene
sulfonic acid (TNBS) [11, 13, 31, 32].
Intraperitoneally injected cryopreserved BMMSCs home to and engraft into the inflamed colon and ameliorate trinitrobenzene
sulfonic acid-induced colitis in rats .
Among various experimental colitis models, hapten-induced colitis in rodents caused by intrarectal administration of trinitrobenzene
sulfonic acid (TNBS) and oxazolone (OXN) is thought of as a type-1 colitis animal model resembling CD and a type-2 model resembling UC, respectively.
In this study a fraction containing the lactone sesquiterpene onopordopicrin (ONP) from Arctium lappa was administered orally as either 25mg/kg daily or 50mg/kg daily 48, 24 and 1 hour before the induction of colitis in rats by the administration of 2,4,6 trinitrobenzene
sulfonic acid (TNBS).
Choi et al., "Catalposide, a compound isolated from Catalpa ovata, attenuates induction of intestinal epithelial proinflammatory gene expression and reduces the severity of trinitrobenzene
sulfonic acid-induced colitis in mice," Inflammatory Bowel Diseases, vol.
Sferra et al., "Smad3 loss confers resistance to the development of trinitrobenzene
sulfonic acid-induced colorectal fibrosis," European Journal of Clinical Investigation, vol.